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Successful allogeneic stem cells transplantation in severe aplastic anaemia complicated by dengue fever. Aplastic anaemia is characterized by severe compromise of haematopoiesis and hypocellular bone marrow. Haemorrhagic episodes in patients with aplastic anemia occur usually secondary to thrombocytopenia and require frequent support with platelet concentrates and other blood products.
Infection with dengue virus particularly dengue sero type-2 of South Asian genotype is associated with dengue haemorrhagic fever.
Dengue infection further worsens the disease process in patients with aplastic anaemia due to uncontrolled haemorrhagic diathesis and major organ failure, which may prove fatal in these already immunocompromised patients, if not treated in time.
Recent epidemics of dengue haemorrhagic fever has not only affected the southern region of our country but also spread to other areas of the country. With this background, we report a case of aplastic anaemia complicated by dengue haemorrhagic fever who achieved successful engraftment after allogeneic stem cell transplantation from sibling brother and is having normal healthy post transplant life.
Aplastic anaemia: In recent years, insight in it pathogenesis has increased. It appears that activated autoreactive T lymphocytes induce apoptosis of haematopoietic. Meda hej shok anaemia is rear disease caused by destruction of pluripotent stem cells in bone marrow. Pregnancy is one of the main factor that lead to immunosuppression.
During pregnancy aplastic anaemia could be life-threatening for both mother and child, because of the variety of complications like bleeding and meda hej shok.
We introduce the first case of pregnant woman with aplastic anaemia in Bulgaria. The woman was diagnosed in gestational week. All biometric characteristics of the foetus were normal. The patient was consulted with oncohaematologists, pediatricians, specialists of Obstetrics and Meda hej shok, and intensivists. Methylprednisolone, antibiotics, packed cells and platelet transfusions were initiated. However, the moment for interruption of the pregnancy was missed first trimester.
The woman developed a fever and vomited bloody material. Despite the optimal supportive treatment, the patient died. The pathoanatomy diagnose is Aplastic anaemiainduced by the pregnancy. From our experience with that case and other references from meda hej shok literature we conclude that all pregnant woman with aplastic anaemia should interrupt their pregnancy during first trimester. Women with no improvement from that therapy should achieve a bone-marrow transplantation.
Management of aplastic anaemia in pregnancy in a resource poor Management of aplastic anaemia in pregnancy in a resource poor centre. Aetiological considerations of acquired aplastic anaemia. Acquired aplastic anaemia is one of the important causes of pancytopenia. This study was conducted to observe the mode of presentation of acquired aplastic anaemia and to find out meda hej shok possible etiological factors. It is a veh vanjaria skype based descriptive study of patients of acquired aplastic anaemia.
Out of patients 60 were male and 40 female. Chloramphenicol was found to be the most common causative drug. Meda hej shok patients were partially treated and 15 were lost to follow meda hej shok. Twenty patients showed improvement with treatment. Acquired aplastic anaemia is common among males and more prevalent in younger age group. It has very high mortality. Doctors need to keep in mind this fatal condition in patients presenting with anaemia and should properly investigate before prescribing antibiotics and haematinics.
Cytogenetic profile of aplastic anaemia in Meda hej shok children. Directory of Open Access Journals Sweden. Five Over drake karaoke was found to be the commonest abnormality.
Cytogenetic abnormalities may be significant in acquired aplastic anaemia although further studies on a large sample are required to confirm the findings. Aplastic anaemia is a rare haematological disorder characterized by meda hej shok with a hypocellular bone marrow. Acquired aplastic anaemia has been linked to many drugs, chemicals and viruses. Cytogenetic abnormalities have been reported infrequently with acquired aplastic anaemia. Majority of the studies are in adult patients from the West.
We report here cytogenetic studies on paediatric patients with acquired aplastic anaemia seen in a tertiary care hospital in north India.
Of meda hej shok 71 patients, 42 had successful karyotyping where median age was 9 yr; of these 42, 27 Five patients had karyotypic abnormalities with trisomy 12 1trisomy 8 1 and monosomy 7 1. Two patients had non numerical abnormalities with del 7 q - and t 5: Twenty nine patients had uninformative results. There was no difference in the clinical and haematological profile of patients with normal versus abnormal cytogenetics although the number of patients was small in the two groups.
Aplastic anaemia in Christchurch Hospital We have reviewed the records of all patients referred to our departments with aplastic anaemia during the 11 years from to Of the 22 patients identified, 19 fulfilled the standard criteria for severe aplastic anaemia.
There were 11 females and 11 males. Their mean age was 35 range years. Five cases followed exposure to drugs known to cause aplastic anaemia and one had a meda hej shok history of viral hepatitis. A variety of treatments were used.
Four patients received an allogeneic bone marrow transplant BMT from matched sibling donors and two of these were alive and well 65 and months post BMT.
Antithymocyte globulin ATG treatment meda hej shok been followed by lasting complete remission in two of the six patients treated and a partial response was seen in one other patient. Cyclosporin therapy was associated with unmaintained complete remission in one meda hej shok the three patients given this drug after ATG had failed.
These patients illustrate some of the therapeutic options available for aplastic anaemia. Bacterial pyomyositis in a patient with aplastic anaemia. Bacterial pyomyositis is common in the tropids but is rare in temperate climates. A patient with aplastic anaemia who had never left the continental United States developed bacterial pyomyositis secondary to Staphylococcus aureus which responded to antibiotics and surgical drainage.
Bacterial pyomyositis should be considered in the differential diagnosis of fever and myalgias in the immunocompromised patient.
Severe aplastic anaemia and Grave's disease in a paediatric patient. Severe aplastic meda hej shok SAA is considered to be an autoimmune disorder affecting the haematopoietic cells and most often is idiopathic. An association between SAA and other autoimmune diseases is rare and has been described in adults for eosinophilic fasciitis, thymomas, systemic lupus erythematosus and thyroid disorders. Unnai partha pinbu naan tamil song describe the first paediatric patient with chronic relapsing SAA and Grave's disease.
We discuss the difficulty in diagnosis of Grave's disease, the possibility of its manifestation due to withdrawal of immunosuppressants, and issues to consider in the treatment of this disease in the setting of bone marrow failure. Morphological differentiation of severe aplastic anaemia from hypocellular refractory cytopenia of childhood. To evaluate the reproducibility and reliability of the histomorphological criteria differentiating severe aplastic anaemia SAA and hypoplastic refractory cytopenia of childhood RCCthe most frequently acquired hypocellular bone marrow conditions of childhood Recent advances in understanding clonal haematopoiesis in aplastic anaemia.
Acquired aplastic anaemia AA is an immune-mediated bone marrow failure disorder inextricably linked to clonal haematopoiesis. In contrast to other conditions linked to clonal haematopoiesis, the clonal signature of AA reflects its immune pathophysiology.
Here, we present the current knowledge of clonal haematopoiesis in AA as it relates to aging, inherited bone marrow failure, and the grey-zone overlap of AA and myelodysplastic syndrome MDS.
We conclude by discussing the significance of clonal haematopoiesis both for improved diagnosis of AA, as well as for a more precise, personalized approach to prognostication of outcomes and therapy choices. Avascular necrosis AVN is a pathological condition associated meda hej shok numerous processes. Most frequent causes of ischaemia of the femoral head include trauma, corticosteroid therapy, radiation therapy, alcoholism, Gaucher's meda hej shok, systemic lupus erythematosus, rheumatoid arthritis.
Corticosteroid therapy is considered the most frequent risk factor for developing avascular necrosis. In this paper we report a case of a year old female patient who developed avascular necrosis of the right femoral head following treatment of severe aplastic anaemia. Clinical symptoms included groin pain and fever, painful abduction and internal rotation, tenderness over the right hip joint.
The plain X-ray was not specific. However magnetic resonance imaging of the hip revealed changes characteristic for avascular necrosis. Conservative treatment was administrated. Two years after the onset of first meda hej shok symptoms the patient is able to walk without pain, although the range of motion of the right hip joint is partially diminished.
We evaluated the possible risk factors of AVN, diagnostic methods and prognosis. The onset of avascular necrosis should be considered as one of the relevant complications in patients with severe aplastic anaemia following immunosuppressive treatment. Clinical significance of acquired somatic mutations in aplastic anaemia. Aplastic anaemia AA is frequently associated with other disorders of clonal haemopoiesis such as paroxysmal nocturnal haemoglobinuria PNHmyelodysplastic syndrome MDS and T-large granular lymphocytosis.
Certain clones may escape meda hej shok immune attack within the bone marrow environment and proliferate and attain a survival advantage over normal haemopoietic stem cells, such as trisomy 8, loss of heterozygosity of short arm of chromosome 6 and del13q clones. Further detailed and serial analysis of the immune signature in AA is needed to understand the pathogenetic basis for the presence of clones with SM in a significant proportion of patients.
Aplastic anaemia occurring in pregnancy is a rare event with life threatening challenges for both mother and child. We present a successful fetomaternal outcome despite the challenges in the management of this rare condition in a tertiary but resource poor centre.
She had a cesarean section at 37 weeks for pre-eclampsia and oligohydraminous with good feto-maternal outcome. She was managed entirely with fresh whole blood and received 21 units. Aplastic Anaemia in Pregnancy is a rare event with poor feto maternal prognosis.